4440666
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A hydrocarbonaceous liquid stream is modified to contain a minor amount of the amide reaction product of a polyalkylene polyamine, a neo-acid having 5 to 20 carbon atoms, the neo-acid having three alkyl groups on the carbon atom which is in the alpha position relative to the carboxylic group, and an aliphatic fatty acid having 10 to 30 carbon atoms, whereby corrosion of iron and other metals contacted by said stream is significantly reduced. The amides of the invention have the structural formula: STR1. Processing Please wait US - Method of making corrosion inhibiting polyamine amides, the amides, and use therefor. Publication Number Publication Date
4440666
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Do you have some more info about this number? A hydrocarbonaceous liquid stream is modified to contain a minor amount of the amide reaction product of a polyalkylene polyamine, 4440666, a neo-acid having 5 to 20 carbon atoms, 4440666, 4440666, the neo-acid having three alkyl groups on the carbon atom which is in 4440666 alpha position relative to the carboxylic group, and an aliphatic fatty acid having 10 4440666 30 carbon atoms, whereby corrosion of iron and other metals contacted by said stream is significantly reduced.
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4440666
Federal government websites often end in. The site is secure. The generation of reactive oxygen species ROS triggered by bacterial endotoxin lipopolysaccharide LPS plays a key role during the pathogenesis of sepsis. We demonstrated that LPS challenge induced marked elevation of lipid peroxidation and nitrite levels in the plasma and tissues of WT mice, as well as elevated pro-inflammatory mediators. In contrast, IRAK-1 deficient mice had significantly lower lipid peroxidation and nitrite levels, as well as lower levels of pro-inflammatory mediators. Additionally, tissues harvested from WT mice injected with LPS exhibited reduced activities of anti-oxidant enzymes including glutathione peroxidase GPx , catalase, and superoxide dismutase SOD. A highly lethal systemic multi-organ dysfunction syndrome MODS can occur following a diverse array of clinical insults, including endotoxemia due to disseminated infection, trauma, and injury Glaros et al. The systemic inflammation that results from these challenges can spread throughout the intravascular space of humans and animals, and ultimately lead to the failure of multiple organ-systems Glaros et al. Antibiotics have been proven ineffective in controlling disseminated endotoxemia, since the bacterial endotoxin lipopolysaccharide LPS , rather than the live bacteria, plays a major role in inducing MODS Bone, LPS alone can trigger multi-organ failure in both humans and laboratory animals Wenzel et al.
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